Frank Brombacher
Frank Brombacher
Verified email at icgeb.org - Homepage
Title
Cited by
Cited by
Year
Requirement for IL-13 independently of IL-4 in experimental asthma
G Grünig, M Warnock, AE Wakil, R Venkayya, F Brombacher, DM Rennick, ...
Science 282 (5397), 2261-2263, 1998
21721998
Macrophage-specific PPARγ controls alternative activation and improves insulin resistance
JI Odegaard, RR Ricardo-Gonzalez, MH Goforth, CR Morel, ...
Nature 447 (7148), 1116-1120, 2007
19542007
A promoter-level mammalian expression atlas
Fantom Consortium
Nature 507 (7493), 462, 2014
11792014
Control of early viral and bacterial distribution and disease by natural antibodies
AF Ochsenbein, T Fehr, C Lutz, M Suter, F Brombacher, H Hengartner, ...
Science 286 (5447), 2156-2159, 1999
9681999
Alternatively activated macrophages produce catecholamines to sustain adaptive thermogenesis
KD Nguyen, Y Qiu, X Cui, YPS Goh, J Mwangi, T David, L Mukundan, ...
Nature 480 (7375), 104-108, 2011
9282011
Tumors induce a subset of inflammatory monocytes with immunosuppressive activity on CD8+ T cells
G Gallina, L Dolcetti, P Serafini, C De Santo, I Marigo, MP Colombo, ...
The Journal of clinical investigation 116 (10), 2777-2790, 2006
8792006
Alternative macrophage activation is essential for survival during schistosomiasis and downmodulates T helper 1 responses and immunopathology
RH De'Broski, C Hölscher, M Mohrs, B Arendse, A Schwegmann, ...
Immunity 20 (5), 623-635, 2004
7682004
Interleukin-17 is a negative regulator of established allergic asthma
S Schnyder-Candrian, D Togbe, I Couillin, I Mercier, F Brombacher, ...
The Journal of experimental medicine 203 (12), 2715-2725, 2006
7362006
IL-5-deficient mice have a developmental defect in CD5+ B-1 cells and lack eosinophilia but have normal antibody and cytotoxic T cell responses
M Kopf, F Brombacher, PD Hodgkin, AJ Ramsay, EA Milbourne, WJ Dai, ...
Immunity 4 (1), 15-24, 1996
6581996
Differential expression of FIZZ1 and Ym1 in alternatively versus classically activated macrophages
G Raes, P De Baetselier, W Noël, A Beschin, F Brombacher, ...
Journal of leukocyte biology 71 (4), 597-602, 2002
5152002
Aluminium hydroxide adjuvant initiates strong antigen-specific Th2 responses in the absence of IL-4-or IL-13-mediated signaling
JM Brewer, M Conacher, CA Hunter, M Mohrs, F Brombacher, ...
The Journal of Immunology 163 (12), 6448-6454, 1999
4971999
IL-25 regulates Th17 function in autoimmune inflammation
MA Kleinschek, AM Owyang, B Joyce-Shaikh, CL Langrish, Y Chen, ...
The Journal of experimental medicine 204 (1), 161-170, 2007
4602007
Transcribed enhancers lead waves of coordinated transcription in transitioning mammalian cells
E Arner, CO Daub, K Vitting-Seerup, R Andersson, B Lilje, F Drabløs, ...
Science 347 (6225), 1010-1014, 2015
4332015
An interleukin 4 (IL-4)-independent pathway for CD4+ T cell IL-4 production is revealed in IL-4 receptor-deficient mice
N Noben-Trauth, LD Shultz, F Brombacher, JF Urban, H Gu, WE Paul
Proceedings of the National Academy of Sciences 94 (20), 10838-10843, 1997
3841997
The transcriptional network that controls growth arrest and differentiation in a human myeloid leukemia cell line
H Suzuki, ARR Forrest, E Van Nimwegen, CO Daub, PJ Balwierz, ...
Nature genetics 41 (5), 553, 2009
3542009
Novel IL-12 family members shed light on the orchestration of Th1 responses
F Brombacher, RA Kastelein, G Alber
Trends in immunology 24 (4), 207-212, 2003
3452003
Sensory neurons co-opt classical immune signaling pathways to mediate chronic itch
LK Oetjen, MR Mack, J Feng, TM Whelan, H Niu, CJ Guo, S Chen, ...
Cell 171 (1), 217-228. e13, 2017
3352017
IL-4-deficient Balb/c mice resist infection with Leishmania major.
M Kopf, F Brombacher, G Köhler, G Kienzle, KH Widmann, K Lefrang, ...
The Journal of experimental medicine 184 (3), 1127-1136, 1996
3341996
IL-4 directly signals tissue-resident macrophages to proliferate beyond homeostatic levels controlled by CSF-1
SJ Jenkins, D Ruckerl, GD Thomas, JP Hewitson, S Duncan, ...
Journal of Experimental Medicine 210 (11), 2477-2491, 2013
3292013
Alternative activation is an innate response to injury that requires CD4+ T cells to be sustained during chronic infection
I Gallagher, MG Nair, X Zang, F Brombacher, M Mohrs, JP Allison, ...
The Journal of Immunology 179 (6), 3926-3936, 2007
3222007
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